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Allostatic hypermetabolic response in PGC1α/β heterozygote mouse despite mitochondrial defects

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dc.contributor.affiliation"Wellcome-MRC Institute of Metabolic Science, University of Cambridge, Cambridge, United Kingdom"," Discovery Sciences, BioPharmaceuticals R&D, AstraZeneca, Gothenburg, Sweden"," VTT, Technical Research Center of Finland, Espoo, Finland"," Department of Physiology, University of the Basque Country UPV/EHU, Bilbao, Spain"," Department of Nutrition, Food Science and Gastronomy, School of Pharmacy and Food Science, Food and Nutrition Torribera Campus, University of Barcelona (UB), Santa Coloma de Gramenet, Spain"," INSA-UB, Nutrition and Food Safety Research Institute, University of Barcelona, Barcelona, Spain"," Department of Cellular and Molecular Physiology, Institute of Translational Medicine, The University of Liverpool, Liverpool, United Kingdom"," School of Science and Technology, Örebro University, Örebro, Sweden"," Institute for Research in Biomedicine (IRB Barcelona), The Barcelona Institute of Science and Technology, Barcelona, Spain"," Dept. Biochemistry and Molecular Biomedicine, University of Barcelona, Barcelona, Spain"," Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas (CIBERDEM), Instituto de Salud Carlos III, Madrid, Spain"," Turku Bioscience Centre, University of Turku and Åbo Akademi University, Turku, Finland"," Research and Early Development Cardiovascular, Renal and Metabolism, BioPharmaceuticals R&D, AstraZeneca, Gothenburg, Sweden"," Division of Endocrinology, Department of Neuroscience and Physiology, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden"," Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Hinxton, United Kingdom"," University of Turku, Turku, Finland"," Madrid Institute for Advanced Studies, Madrid, Spain"," Novo Nordisk, Copenhagen, Denmark"en
dc.contributor.affiliation000000041762408X
dc.contributor.authorRodriguez-Cuenca S.
dc.contributor.authorLelliot C.J.
dc.contributor.authorCampbell M.
dc.contributor.authorPeddinti G.
dc.contributor.authorMartinez-Uña M.
dc.contributor.authorIngvorsen C.
dc.contributor.authorDias A.R.
dc.contributor.authorRelat J.
dc.contributor.authorMora S.
dc.contributor.authorHyötyläinen T.
dc.contributor.authorZorzano A.
dc.contributor.authorOrešič M.
dc.contributor.authorBjursell M.
dc.contributor.authorBohlooly-Y M.
dc.contributor.authorLindén D.
dc.contributor.authorVidal-Puig A.
dc.date.accessioned2021-10-04T11:18:02Z
dc.date.available2021-10-04T11:18:02Z
dc.date.issued2021
dc.identifier.doi10.1096/fj.202100262RR
dc.identifier.urihttp://hdl.handle.net/20.500.12614/2708
dc.issue.numbere21752
dc.journal.titleFASEB Journal
dc.language.isoen
dc.relation.projectIDinfo:eu-repo/grantAgreement/EC/FP7/223450/EU/INTEGRATION OF THE SYSTEM MODELS OF INSULIN SIGNALLING AND OF MITOCHONDRIAL FUNCTION AND ITS APPLICATION IN THE STUDY OF COMPLEX DISEASES/MITIN
dc.relation.projectIDinfo:eu-repo/grantAgreement/WT/////
dc.relation.projectIDTEMP"This work was funded by FP7‐MITIN [HEALTH‐F4‐2008‐223450] and the MRC MDU [MC_UU_12012/2]. SRC was also funded by MEIF‐CT‐2005‐023061. The Biochemistry Assay Lab and the Histopathology Core are funded by MRC Metabolic Diseases Unit [MC_UU_00014/5]"," Imaging Core is funded by Wellcome Trust Major Award [208363/Z/17/Z]."
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 España*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/3.0/es/*
dc.titleAllostatic hypermetabolic response in PGC1α/β heterozygote mouse despite mitochondrial defects
dc.typeresearch article
dc.volume.number35
dspace.entity.typePublication

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